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Based on our data, we propose that T3 has a regulatory role in insulin-controlled glucose metabolism, and T3 exerts this function by increasing non-12α-OH FXR-antagonistic BA levels via downregulating the expression of CYP8B1, which potentiates GLP-1 production via suppressing the intestinal FXR signaling
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Clinicians should take a thorough cardiovascular history and record a baseline heart rate before initiating treatment, and reassess regularly throughout the course of therapy
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