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These categories included genomic instability, telomere attrition, epigenetic alterations, loss of proteostasis, deregulated nutrient sensing, mitochondrial dysfunction, cellular senescence, stem cell exhaustion, and altered intercellular communication [2]
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[1] [2] These agents work by mimicking the action of the naturally occurring incretin hormone GLP-1, which is released from the intestine in response to food intake
