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Furthermore, compelling evidence implicates this local, brainstem-derived GLP-1, rather than gut-derived GLP-1, as a principal modulator of physiological GLP-1R dependent signals that regulate appetite and body weight (Drucker and Asa, 1988
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They used inverse probability-weighted Cox survival models to estimate the hazard ratios (HRs) and three-year net risk differences (NRD) per 1,000 individuals for statistical analysis
(1)These statements have not been evaluated by the Food and Drug Administration
